<div style=”color: rgb(0, 0, 0); font-family: Verdana, Arial, Helvetica, sans-serif; font-size: 10px; margin-top: 0px; margin-right: 0px; margin-bottom: 0px; margin-left: 0px; background-image: initial; background-attachment: initial; background-origin: initial; background-clip: initial; background-color: rgb(255, 255, 255); font: normal normal normal 13px/19px Georgia, ‘Times New Roman’, ‘Bitstream Charter’, Times, serif; padding-top: 0.6em; padding-right: 0.6em; padding-bottom: 0.6em; padding-left: 0.6em; “><p>Bioimpendance Neuronal Micro-stimulation (BNM) was engineered to treat sports injury and muscle atrophy, as well as promote lipolysis and muscle hypertrophy. A clinical study with individuals presenting abnormally clumped RBCs was completed in February 2009 with the Ion Magnum. Results indicate that this technology rapidly and efficiently leads to normalized erythrocytes’ separation at the microscopic level. Red Blood Cells’(RBCs) separation is crucial for the timely transport of hormones,antibodies, oxygen and nutrients to the cells and waste products to the kidneys. Therefore, blood separation is crucial in a number of biological processes including cellular cleansing, nourishment and oxygenation, endocrine and immune functioning. Ion Magnum’s (IM) dynamic, multi-sine, analogue waveform was originally tested at the cellular level by Dr. Donald Gilbert (1992), a molecular biologist, and was electronically composed by the Co-Inventor of the first Pacemaker (2008) to resonate at the motor nerve the signal of strenuous exercise normally emitted by the brain. Due to its resonance with the biological signal, the Ion Magnum signal spreads throughout the CNS ultimately triggering hormonal secretion such as Growth Hormone (GF), Thyroxine (T4) and Triiodothyronine (T3) for lipolysis and Insulin Growth Factor (IGF-1) for muscle hypertrophy. Power detox is an additional benefit of Ion Magnum’s induced effortless and painless isometric and isotonic muscle contractions. Sever al devices stimulate the muscles such asTENS Muscle Stimulators. However, TENS devices deplete ATP. Besides, muscle stimulation does not automatically release hormones. Neuronal synapses activated out of sync with the other inputs to the neuron stands out as odd and are eliminated. Neuronal synapses that are activated in sync with other inputs to the neuron are strengthened. The signal of a device must be in sync with the biological choreography in order to spread via the spinal cord and reach the brain. In sync, or resonance, has been touted by a number of approximate hit-ormiss techniques involving magnetic and electrical fields with dubious inconsistent results. But no technology has ever achieved nerve signaling that is biologically comparable to physical exercise. What the Co-Inventor of the first Pacemaker has accomplished, first in London University and then in the EU funded Research Center for Innovations Science, UK, is a full force, high-speed workout without actual movement, side effects or pain, that enhances hormonal secretion. Lipolysis and muscle hypertrophy following IM treatments has been reported by a number single subject design clinical studies.
March 27, 2011
Healthy Blood Vessels May Prevent Fat Growth. Xanya Sofra Weiss
<span class=”Apple-style-span” style=”font-family: Georgia, ‘Times New Roman’, ‘Bitstream Charter’, Times, serif; font-size: 13px; line-height: 19px; “><p>ScienceDaily (Sep. 23, 2008) — The cells lining blood vessels are known to be important for maintaining health, but researchers at the Indiana University School of Medicine believe these cells may perform an unsuspected task – controlling the development of fat cells.</p><p>Their findings are reported in the September issue of the journal Stem Cells.</p><p>The researchers found that precursor or stem cells have a markedly reduced tendency to develop into fat cells when placed in direct contact with healthy endothelial cells, which are the cells that line blood vessels.</p><p>”The key to this discovery was our recent observation that these cells, also known as adipose stromal cells, in fat tissue are in very close contact with endothelial cells in small blood vessels and capillaries,” said Keith L. March, M.D., Ph.D., co-principal investigator of the study and director of the Indiana Center for Vascular Biology and Medicine (ICVBM).</p><p>”Once we had recognized this link between endothelial and stromal cells, it was a logical step to ask how these cells can influence each other,” said Dr. March, who also is a professor of medicine, physiology and biomedical engineering at the IU School of Medicine and Krannert Institute of Cardiology.</p><p>Dr. March and his colleagues are researching uses for one of nature’s building blocks, adipose stem cells, which they harvest from fat tissues. Their research looks at ways to treat vascular disease, including the use of adipose stem cells to grow new vessels as a treatment for peripheral artery disease.</p><p>When the adipose stem cells were mixed with endothelial cells, they were less likely to develop into fat cells, said Gangaraju Rajashekhar, Ph.D., lead author of the journal article and a research associate at the Indiana Center for Vascular Biology and Medicine.</p><p>What we discovered was that endothelial cells released proteins – including Wnt proteins to be precise – that play a significant role in blocking fat cell development. Wnt proteins regulate development and differentiation in many tissues and may even play a role in aging,” said Dr. Rajashekhar.</p><p>The researchers cautioned that more studies are needed to determine whether repair of unhealthy endothelial cells also can help to control fat growth.</p><p>”We know now that endothelial cells in blood vessels cells help tell adipose stem cells what to do,” said Matthias Clauss, Ph.D., an ICVBM co-principal investigator of the study and an associate professor for cellular and integrative physiology at the IU School of Medicine.</p><p>”What we don’t know yet is how the formation of fat cells influences the blood vessel lining cells. Our current hypothesis is that endothelial dysfunction promotes fat cell development, accompanied by new blood vessel growth. We hope to soon be able to interrupt this cycle,” said Dr. Clauss.</p><p>This research may result in new options for treatment of cardiovascular diseases and also could provide physicians with another weapon in the war on obesity.</p><p></p><p><a href=”http://xanya-sofra-weiss.palaweb.com/info-xanya_sofra_weiss-nanotechnology” mce_href=”http://xanya-sofra-weiss.palaweb.com/info-xanya_sofra_weiss-nanotechnology”>Xanya Sofra Weiss</a></p><p>Xanya Sofra Weiss</p><p></p></span>
Using Quantum Medicine to Unravel Stressors That Provoke Carcinogenesis. Xanya Sofra Weiss
<div style=”color: rgb(0, 0, 0); font-family: Verdana, Arial, Helvetica, sans-serif; font-size: 10px; margin-top: 0px; margin-right: 0px; margin-bottom: 0px; margin-left: 0px; background-image: initial; background-attachment: initial; background-origin: initial; background-clip: initial; background-color: rgb(255, 255, 255); font: normal normal normal 13px/19px Georgia, ‘Times New Roman’, ‘Bitstream Charter’, Times, serif; padding-top: 0.6em; padding-right: 0.6em; padding-bottom: 0.6em; padding-left: 0.6em; “><div>Quantum physics when applied to the study of biological processes is known as biophysics. Biophysics studies the living cell as a whole system with electrical fields that interrelate and penetrate the entire organism.1 Carcinogenesis occurs when healthy bioelectrical fields are transformed. Quantum Medicine applies quantum physics theories to unravel the stressors that cause disruptions within bioelectrical fields.</div><div></div><div></div><div><div></div><div><a href=”http://xanya-sofra-weiss.palaweb.com/info-xanya_sofra_weiss-nanotechnology” mce_href=”http://xanya-sofra-weiss.palaweb.com/info-xanya_sofra_weiss-nanotechnology”>Xanya Sofra Weiss</a></div><div></div><div>Xanya Sofra Weiss</div></div></div>
Human Adipose Tissue Blood Flow and Micromanipulation of Human Subcutaneous Blood Flow. Xanya Sofra Weiss
Xanya Sofra Weiss
Diabetic Peripheral Neuropathy. Xanya Sofra Weiss
O B J E C T I V E— To evaluate the efficacy of combining electrotherapy with amitriptyline for the management of chronic painful peripheral neuropathy in patients with type 2 diabetes. RESEARCH DESIGN AND METHODS— Patients (n = 26) with peripheral neuro p athy were treated with amitriptyline. After 4 weeks, those patients (n = 23) who failed to re s p o n d to amitriptyline or who only had partial relief were randomized between a sham tre a t m e n t g roup (control) or an electrotherapy group. Transcutaneous electrotherapy was given for 12 weeks by a portable unit (H-wave machine) that generated a biphasic exponentially decaying w a v e f o rm (pulse width 4 ms, 25–35 V, 2 Hz). The degree of pain and discomfort was graded on a scale of 0–5. An analog scale was used to re c o rd the overall change in symptoms.
R E S U LT S— Amitriptyline produced some degree of symptomatic relief in 15 (60%) of the 26 patients by the 4th week; pain scores decreased from 3.8 ± 0.1 to 2.9 ± 0.2 (P 0.1) and the overall reduction in pain was 26 ± 5% on an analog scale. In the amitriptyline plus sham treatment group (n = 9), pain scores declined from 2.8 ± 0.3 to 1.9 ± 0.5 (P 0.03) and the overall reduction in pain was 55 ± 12%, suggesting a pro c e d u re – related placebo effect. In the group receiving combined electrotherapy and amitriptyline (n = 14), symptomatic impro v ement occurred in 12 (85%) patients. Five (36%) of the patients in this group became asymptomatic. Pain scores declined from 3.2 ± 0.2 to 1.4 ± 0.4 (P 0.01) and the overall re d u c t i o n in pain was 66 ± 10%. The degree of reduction in pain scores and the incremental relief (above the amitriptyline effect) were significantly greater (P 0.03) with electrotherapy as compare d with sham treatment. The outcomes indicate a substantial beneficial effect of electro t h e r a p y over and above any placebo influ e n c e . C O N C L U S I O N S— Our clinical observations suggest that transcutaneous electrotherapy is effective in reducing the pain associated with peripheral neuro p a t h y. This form of therapy may be a useful adjunctive modality when it is combined with a pharmacological agent, such as amitriptyline, to augment symptomatic re l i e f .
Xanya Sofra Weiss